The Prime Cause and Prevention
of Cancer
From the 1966 Lindau Nobel-Laureates Conference, Germany Presented by
Otto Warburg, Nobel Prize-winner
What you are about to read may shock you. You may become upset to learn
that this information has not been publicized. Professor
Warburg’s conclusions are virtually identical to the conclusions reached
by today’s science of Life -Systems Engineering.
The text is technical, but the footnotes explain the logic and key
issues.
But, even for cancer, there is only one main cause. Summarized
in a few words, the prime cause of cancer is the
replacement of the respiration of oxygen in normal body cells by
a fermentation of sugar.
It has been shown if the active groups of the respiratory enzymes
are removed from food cells, respiration is impaired; and that
cell respiration is repaired at once, if these groups are
again added to food.”
Neither genetic codes of anaerobiosis nor cancer viruses are
alternatives today, because no such codes and no such viruses in man
have been discovered so far …
The first active group of an enzyme, ‘Iron, the
Oxygen-Transferring Part of the Respiratory Enzyme’ was discovered in
1924.
Furthermore, the very unexpected and fundamental fact
that tissue culture is carcinogenic [cancer-causing] and
that a too low oxygen pressure is the intrinsic (prime)
cause of cancer was discovered in the years 1927-1966.
Because no cancer cell exists, the respiration of which is intact, it
cannot be disputed that cancer could be prevented if the respiration of
the body cells would be kept intact. Today we know two methods to
influence cell respiration.
The first is to decrease the oxygen pressure in growing cells.
If it is so much decreased that the oxygen transferring enzymes are
no longer saturated with oxygen, respiration can decrease
irreversibly and normal cells can be transformed into
facultative anaerobes.
The second method to influence cell respiration in the body is to add
the active groups of the respiratory enzymes to the food of man.
Lack of these groups impairs cell respiration and
abundance repairs impaired cell respiration.
To prevent cancer it is therefore proposed to keep the speed
of the blood stream
so high that the venous blood still contains sufficient oxygen; second,
to keep high concentrations of hemoglobin
in the blood; third to add always to food, even of healthy
people, the active groups of the respiratory enzymes;
and to increase the doses of these groups, if a precancerous state has
already developed. If at the same time exogenous carcinogens
are excluded rigorously.
Cancer, above all other diseases, has countless secondary causes. But,
even for cancer, there is only one main cause.
Summarized in a few words, the prime cause of cancer is the
replacement of the respiration of oxygen in normal body
cells by a fermentation of sugar. All normal cells
meet their energy needs by the respiration of oxygen, whereas
cancer cells meet their energy needs in great part by
fermentation. From the standpoint of chemistry and physics of
life this difference between normal and cancer cells is so great
that one can scarcely picture a greater difference. Oxygen
gas, the donor of energy in plants and animals is dethroned
in the cancer cells and replaced by an energy yielding reaction
of the lowest forms, namely a fermentation of glucose [sugar].”
… cancer cells can actually grow in the body almost with only the
energy of fermentation.
In the mouse,
if one provides an oxygen pressure so reduced that the oxygen
respiration is partially inhibited, the purely aerobic metabolism of the
mouse embryonic cells is quantitatively altered in 48 hours in
the course of two cell divisions. If one increases to the
original high oxygen pressure, and allows the cell to grow further,
the cancer metabolism remains, it’s an irreversible process.
We find by experiment about 35% (about 1/3rd less)
inhibition of oxygen respiration already suffices to bring about
such a transformation during cell growth.
In any case, during the cancer development the oxygen-respiration
always falls, fermentation appears, and the highly
differentiated cells are transformed to fermenting aerobes, which have
lost all their body functions and retain only the now useless property
of growth…. The meaning of life disappears.
The endproducts of fermentation are reached by one single reaction. On
the other hand, the endproducts of the oxidation of pyruvic acid are
only reached after many additional reactions. Therefore, when cells are
harmed, it is probable that first respiration is harmed.”
Now, don’t shoot me – I’m only the
messenger. But you may want to shoot someone as you read on and realize
that these anti-cancer medical articles have been published, but have
been kept hidden from you! Here are just a few of the significant
articles written between 1985 – 1999 stressing the anti-cancer
properties of basic essence healthy essential oils:
Effect of essential fatty acids on natural cytotoxicity in patients
with colorectal cancer
European Journal of Surgical
Oncology (cancer)1995
June;21(3):254-260
ˇ
“Essential fatty acids (basic essence) have
been shown to modulate [control] lymphocyte reactivity and
destroy various cancer cells in vitro (in a test tube).
ˇ
“… NK cell activity showed no significant change
following 15 days ingestion of EFAs in the group with localized
cancer, but was significantly reduced in the group with
advanced [cancer] disease and continued to decline reaching
minimal levels following 6 months supplementation….”
ˇ
“More importantly, the cytotoxicity of NK and LAK
cells returned to the pre-supplementation values, 3 months after
cessation of EFA intake!”
Dietary deficiency or enrichment of essential fatty acids modulates
[controls] tumorigenesis [tumorous cells] in the whole body of
cobolt-60-irradiated mice
Prostaglandins Leukot
Essential Fatty Acids 1997
March;56(3):239-244
ˇ
“It may be concluded that, when a tumor
initiator injures the body as a whole, EFAD (basic essence
deficiency) achieved either through a fat-free or
oleic-supplemented (non-EFA, like olive oil) behaves as a general
promoting condition for tumorigenesis.”
Gamma linolenic acid regulates expression of maspin and the
motility of cancer cells
Biochemistry and Biophysics Research Commun 1997 Aug
28;237(3):639-644
ˇ
“Maspin is a recently identified tumour suppressor and
has a profound effect on cell motility…. It is concluded that GLA,
a member of the omega 6 series EFAs [found in basic essence],
up-regulates the expression of maspin which is associated with
a reduction in the motility (growth) of cancer cells [less
cancer].”
Is the risk of urinary tract tumorigenesis enhanced by a marginal
chronic essential fatty acid deficiency (EFAD)?
Nutrition 1998 Feb;14(2):211-216
ˇ
“A considerable amount of experimental,
clinical, and epidemiological data indicates that dietary
fats play a role in urinary tract tumorigenisis.
ˇ
“…In other cell populations, EFAs [basic essence]
are beneficial as preventative and therapeutic nutrients for
treatment of cancer.”
Regulation of cell adhesion, a central mechanism in the anticancer
action of essential fatty acids
International Journal of Medicine 1998 March;1(3):621-625
ˇ
“Certain members of essential fatty acids (basic
essence EFAs) are known to have anticancer functions.
ˇ
“…established in recent years that some of
these anticancer properties are via the effects of these
fatty acids on the adhesive properties of cancer cells,
including cell-cell and cell-matrix adhesions.
ˇ
“…EFAs [basic essence] as a group of novel (unusual)
agents, able to regulate adhesive function of cancer cells
and thus bearing clinical importance.”
Eicosatrienoic acid (20:3 n-9) inhibits the expression of E-cadherin
and desmoglein in human squamous carcinoma in vitro
Prostaglandins Leukot Essential Fatty Acids 1998
December;59(6):371-377
ˇ
“Eicosatrienoic acid is abnormally increased by
essential fatty acid [basic essence deficiency], a
condition associated with alterations of cell proliferation
and differentiation.”
Dynamics of reactive oxygen intermediate production in human glioma:
n-6 essential fatty acid effects
European Journal of Clinical Investigation 1999
March;29(3):220-223
ˇ
“Reactive oxygen intermediates (ROIs) are important
signals controlling cell growth and cell death. Local
essential fatty acid [EFA] deficiencies in tumour cells
may limit ROI generation. This deficiency may be
rectified by addition of exogenous EFA [basic
essence].”
ˇ
“…The stimulated [by basic essence EFAs]
ROI rate was exponential [very significant].”
Dietary fats and cancer
Medical Hypotheses 1985 August;17(4):351-362
ˇ
“The mono-unsaturated non-essential fatty acid
oleic acid (OA) [primary fat in olive oil] has been shown to
stimulate malignant cell proliferation in culture
significantly.
In contrast, the essential fatty acids [EFAs] and their
derivatives have been shown to have potent
proliferation suppressive [stops growth] effects on malignant
[cancerous] cells in culture.”
ˇ
“It has been repeatedly demonstrated that EFAs
and EFA metabolites suppress proliferation of a wide range of
malignant cells in culture.”
ˇ
“…The evidence
reviewed by committee suggests that cancers of most of the major
sites are influenced by dietary pattern….is most suggestive for a
causal relationship between fat intake and the occurrence of
cancer.”
Can
linoleic acid and gamma-linolenic acid be important in cancer
treatment?
Medical
Hypotheses
1994
December;43(6):415-417
ˇ
“…Addition
of LA and GLA [basic
essence] to
available therapeutic regimens may be
worth considering in cancer treatment.”
Life-Systems Engineering analysis: As early as 1985 it has
been published in the world’s leading medical journals that a
significant nutritional deficiency significantly increases the odds of
contracting cancer. It was published in 2001 that cancer has little
genetic basis, but few listened. Nobel Prize-winning Professor Otto
Warburg’s work (and warning) of 1966 has proven itself 100%.
Why have you not been told of this easy
solution to prevent cancer?
Analysis shows: it’s proteins, not genes, that count
— Reuters Science News, February 11, 2001.
1. “Both teams
agree: it is proteins that matter—much more than genes.”
2. “Genes
don’t determine whether you get colon cancer …”